References

Allen WR, Bracher V. Videoendoscopic evaluation of the mare's uterus: III. Findings in the pregnant mare. Equine Vet J. 1992; 24:(4)285-291 https://doi.org/10.1111/j.2042-3306.1992.tb02837.x.tb02837.x

Bidwell LA. Anesthesia for dystocia and anesthesia of the equine neonate. Vet Clin North Am Equine Pract. 2013; 29:(1)215-222 https://doi.org/10.1016/j.cveq.2012.11.003

Busse NI, Uberti B. Uterine Inertia due to Severe Selenium Deficiency in a Parturient Mare. J Equine Vet Sci. 2020; 85 https://doi.org/10.1016/j.jevs.2019.102845

Byron CR, Embertson RM, Bernard WV Dystocia in a referral hospital setting: approach and results. Equine Vet J. 2003; 35:(1)82-85 https://doi.org/10.2746/042516403775467405

Card CE, Wood MR. Effects of Acute Administration of Clenbuterol on Uterine Tone and Equine Fetal and Maternal Heart Rates1. Biology of Reproduction. 1995s; 52:(monograph_series1)7-11 https://doi.org/10.1093/biolreprod/52.monograph_series1.7

Corley KTT, Axon JE. Resuscitation and emergency management for neonatal foals. Vet Clin North Am Equine Pract. 2005; 21:(2)431-455 https://doi.org/10.1016/j.cveq.2005.04.010

Davies Morel MCG, Newcombe JR, Holland SJ. Factors affecting gestation length in the Thoroughbred mare. Anim Reprod Sci. 2002; 74:(3-4)175-185 https://doi.org/10.1016/S0378-4320(02)00171-9

Erkert RS, Macallister CG. Isoxsuprine hydrochloride in the horse: a review. J Vet Pharmacol Ther. 2002; 25:(2)81-87 https://doi.org/10.1046/j.1365-2885.2002.00386.x

Frazer GS, Perkins NR, Blanchard TL Prevalence of fetal maldispositions in equine referral hospital dystocias. Equine Vet J. 1997; 29:(2)111-116 https://doi.org/10.1111/j.2042-3306.1997.tb01651.x

Frazer GS. Chapter 59: Dystocia and fetotomy. In: Samper JC, Pycock JF, McKinnon AO (eds). Saint Louis: WB Saunders; 2007 https://doi.org/10.1016/B978-0-7216-0252-3.50063-1

Frazer G. Dystocia management. In: McKinnon AO, Squires EL, Vaala WE, Varner DD (eds). New Delhi: Wiley-Blackwell; 2011a

Frazer G. Fetotomy. In: McKinnon AO, Squires EL, Vaala WE, Varner DD (eds). New Delhi: Wiley-Blackwell; 2011b

Giles RC, Donahue JM, Hong CB Causes of abortion, stillbirth, and perinatal death in horses: 3,527 cases (1986–1991). J Am Vet Med Assoc. 1993; 203:(8)1170-1175

Ginther OJ, Williams D. On-the-farm incidence and nature of equine dystocias. J Equine Vet Sci. 1996; 16:(4)159-164 https://doi.org/10.1016/S07370806(96)80131-6

Ginther OJ. Equine pregnancy: physical interactions between the uterus and conceptus. AAEP Proceedings. 1998; 44:73-104

Grubb TL, Riebold TW, Huber MJ. Comparison of lidocaine, xylazine, and xylazine/lidocaine for caudal epidural analgesia in horses. J Am Vet Med Assoc. 1992; 201:(8)1187-1190

Lu KG, Barr BS, Embertson R Dystocia—A True Equine Emergency. Clinical Techniques in Equine Practice. 2006; 5:(2)145-153 https://doi.org/10.1053/j.ctep.2006.03.008

Luukkanen L, Katila T, Koskinen E. Some effects of multiple administrations of detomidine during the last trimester of equine pregnancy. Equine Vet J. 1997; 29:(5)400-403 https://doi.org/10.1111/j.2042-3306.1997.tb03147.x

McCue PM, Ferris RA. Parturition, dystocia and foal survival: a retrospective study of 1047 births. Equine Vet J. 2012; 44:(41)22-25 https://doi.org/10.1111/j.2042-3306.2011.00476.x

Squires EL, Hughes SE, Ball BA Effect of season and reproductive status on the incidence of equine dystocia. J Equine Vet Sci. 2013; 33:(5) https://doi.org/10.1016/j.jevs.2013.03.123

Stanton ME. Uterine involution. In: McKinnon AO, Squires EL, Vaala WE, Varner DD (eds). New Delhi: Wiley-Blackwell; 2011

Turner RM. Post-partum problems: the top ten list. American Association of Equine Practitioners. 2007; 53:305-319

Vandeplassche MM. The pathogenesis of dystocia and fetal malformation in the horse. J Reprod Fertil Suppl. 1987; 35:547-552

Vandeplassche M, Spincemaille J, Bouters R Some aspects of equine obstetrics. Equine Vet J. 1972; 4:105-113

Vasey JR, Russell T. Uterine torsion. In: McKinnon AO, Squires EL, Vaala WE, Varner DD (eds). New Delhi: Wiley-Blackwell; 2011

Understanding dystocia in the field: part 1

02 March 2021
13 mins read
Volume 5 · Issue 2
Figure 1. Dystocia caused by ventrally deviated head and neck. The foetal poll was palpable and the muzzle was retrieved and guided into the pelvic inlet, allowing an uncomplicated assisted vaginal delivery. Although born alive, the foal was euthanised because of congenital deviation of the rostral maxilla and nasal septum (wry-nose).
Figure 1. Dystocia caused by ventrally deviated head and neck. The foetal poll was palpable and the muzzle was retrieved and guided into the pelvic inlet, allowing an uncomplicated assisted vaginal delivery. Although born alive, the foal was euthanised because of congenital deviation of the rostral maxilla and nasal septum (wry-nose).

Abstract

Difficult births (dystocias) are not uncommon for horses and present significant risks to mare and foal, with potentially fatal consequences. A sound understanding of the normal physiological processes before and during parturition are essential to enable vets to quickly identify when these processes deviate from the norm. To rapidly diagnose the cause of the dystocia, the attending obstetrician should be assured in their approach; resolute in their decision making; and prompt in action if the risks of mare and foal morbidity and mortality are to be managed. Constructive communication with the mare's owners is essential to allow adequate planning to prioritise the outcome. The future fertility of the mare is imperative in many cases yet is often overlooked. The obstetrician must be dynamic and be prepared to alter their approach when necessary. This review outlines the causes of dystocia and the recommended approaches to resolution.

Dystocia in equines can be life-threatening to both dam and foetus, and represents a genuine emergency. Dystocia is reported to affect between 1–13.2% of foalings, with large Draft and pony breeds considered to be at higher risk than other breeds (Ginther and Williams, 1996; Lu et al 2006; Frazer, 2007). Dystocia was found to be less likely in maiden Thoroughbred mares (8.5%) than barren mares (12.7%) or mares that had previously foaled (14.2%) (Squires et al, 2013). Regardless of breed or parity, it is recommended that all equine births are attended so that if manual intervention is required, it can be provided in a timely fashion.

From approximately 2 months into gestation, the equine foetus is highly active (Allen and Bracher, 1992). The characteristically long umbilical cord facilitates free movement within the allantoic cavity between the uterine body and gravid horn, and alterations in foetal polarity between cranial and caudal presentation occur regularly. Between 5–7 months, both horns contract and the foetus is confined to the uterine body. By 7 months, the uterine horns form an acute angle over the dorsal aspect of the markedly enlarged uterine body. The foetus' hindfeet regain access to the gravid uterine horn, which is only possible while in cranial presentation and dorsal recumbency (Ginther, 1998). The hind-feet advance through the horn lumen with continued growth, to reach the horn tip by 10 months. Once the hindlimbs are enclosed within the gravid horn, the foetus is committed to a cranial longitudinal presentation. These uterofoetal interactions ensure that the vast majority (98.9%) of births occur in cranial longitudinal presentation; with only 1.0% in caudal longitudinal presentation and 0.1% in transverse presentation (Vandeplassche, 1987). Significant episodic foetal activity continues to term, which can be vigorous and sometimes clearly visible through the mare's flank. The soft eponychium extending from the pointed foetal hooves is thought to provide some cushioning to protect the uterus from potential injury.

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